ATG9A Proteins

ATG9A (Autophagy Related 9A) is a Protein Coding gene. Diseases associated with ATG9A include Age-Related Hearing Loss and Neurodegeneration With Brain Iron Accumulation 5. Among its related pathways are Autophagy - animal and Autophagy Pathway. Gene Ontology (GO) annotations related to this gene include heme-copper terminal oxidase activity. An important paralog of this gene is ATG9B.

The following recombinant ATG9A proteins are manufactured in house under a complete QC system by CUSABIO. They are expressed by Yeast, E.coli, Baculovirus, Mammalian cell, In Vivo Biotinylation in E.coli. Highlights of these recombinant ATG9A proteins as follow:
High purity, Low endotoxin, Multiple Tags, Animal-free, Wide applications (Cell assay, Protein-protein interaction, Drug-related studies, Enzymatic activity in vitro, Protein structure analysis, etc.)
In addition, various options on sizes, excellent technical support, and recombinant ATG9A proteins custom service will be also offered.

ATG9A Proteins Catalog

ATG9A Proteins for Mus musculus (Mouse)

ATG9A Proteins for Bos taurus (Bovine)

ATG9A Proteins for Rattus norvegicus (Rat)

ATG9A Proteins for Pongo abelii (Sumatran orangutan) (Pongo pygmaeus abelii)

ATG9A Proteins for Homo sapiens (Human)

ATG9A Background

Autophagy-related protein 9A is a protein in humans that is encoded by the ATG9A gene. The ATG9A protein is involved in autophagy and cytoplasm to vacuole transport (Cvt) vesicle formation. It plays a key role in the organization of the pre-autophagosomal structure/phagophore assembly site (PAS), the nucleating site for the formation of the sequestering vesicle. ATG9A cycles between a juxta-nuclear trans-Golgi network compartment and late endosomes. Nutrient starvation induces accumulation on autophagosomes. Starvation-dependent trafficking requires ULK1, ATG13, and SUPT20H.

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