eve Antibody

1. Pericardial cells that express the transcription factor Even Skipped adopt distinct fates along the anterior-posterior axis. Among them, the most anterior Antp-Ubx-AbdA-negative cells form a novel cardiac outflow component we call the outflow hanging structure, whereas the Antp-expressing cells differentiate into wing heart precursors. PMID: 29247145
2. natural variation in a conserved trait-even-skipped (eve) expression at the cellular blastoderm stage of embryonic development in Drosophila melanogaster, was investigated. PMID: 26129990
3. Ubiquitous TER94 expression is "replaced" by expression in an eve pattern when Homie is deleted, and this effect is reversed when the PRE is also removed PMID: 24204298
4. Deletion of any individual enhancer reduced Yan occupancy at the other elements, impacting eve expression, cell fate specification, and cardiac function PMID: 24186975
5. Data strongly suggests that the eve-grn transcriptional code controls axon guidance, in part, by regulating the level of unc-5 expression. PMID: 22461564
6. even-skipped genes were misexpressed in pho mutant embryos PMID: 22080372
7. Nmo regulates a subset of Eve activities by stimulating Eve-mediated suppression of the odd-skipped (odd) repressor PMID: 20398650
8. The repressor activity of Even-skipped is highly conserved, and is sufficient to activate engrailed and to regulate both the spacing and stability of parasegment boundaries. PMID: 12223400
9. three distinct mechanisms are required for anterior repression of a single eve enhancer, each in a specific position PMID: 12397102
10. the eve enhancer integrates multiple positive and negative transcription factor inputs to restrict eve expression to a single precursor cell and its motoneuron progeny PMID: 14556285
11. opposing gradients of two Drosophila transcriptional repressors, Hunchback (Hb) and Knirps (Kni), position several segments by differentially repressing two distinct regulatory regions (enhancers) of the pair-rule gene even-skipped (eve) PMID: 14685241
12. The pair-rule segmentation gene even skipped (eve) is required to activate engrailed stripes and to organize odd-numbered parasegments (PSs). PMID: 15110723
13. Functional genetic complementation analysis of a eukaryotic cis-regulatory module-the even-skipped stripe 2 enhancer-from four Drosophila species. PMID: 15757364
14. A high-throughput method allows for the quantification of eve expression in early embryos. PMID: 15834586
15. The homeobox transcription factor even-skipped regulates netrin-receptor expression to control dorsal motor-axon projections in Drosophila. PMID: 16085495
16. in developing Drosophila cardiac progenitor cells, the Eve precursors next to the Hh stripe are distinguished from more distant Lbe precursors by locally augmenting Ras signaling via elevating rho transcripts PMID: 16387294
17. An eve-->grn-->zfh1 genetic cascade is unique to one of the intersegmental motoneurons, the aCC. PMID: 16540509
18. A model of the transcriptional readout of the proximal 1.7 kb of the control region of the Drosophila melanogaster gene even skipped (eve). PMID: 16980977
19. Eve is sufficient to regulate both voltage- and ligand-gated currents in motoneurons, extending its known repertoire of action beyond its already characterized role in axon guidance. PMID: 17147779
20. Spatial specificity of mesodermal eve expression relies on multiple repressor sites. PMID: 18067885
21. The sepsid and Drosophila enhancers drive nearly identical expression patterns in transgenic D. melanogaster embryos. PMID: 18584029
22. the single ortholog of Dbx1/2 contributes to the development of specific subsets of interneurons via cross-repressive, lineage-specific interactions with the motoneuron-promoting factors eve and hb9 (exex) PMID: 19710170

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KEGG: dme:Dmel_CG2328

STRING: 7227.FBpp0087478

UniGene: Dm.18510