CaMKII Antibody

1. It is NMDA receptor downstream kinase and essential for memory and learning acquisition.(review) PMID: 23649390
2. Regulated CaMKII autophosphorylation appears essential in central synapse development, and particular cholinergic synapses are affected differentially, although they operate via the same nicotinic receptor. PMID: 22219280
3. These results suggest that presynaptic CaMKII activation depends on autophosphorylation and global Ca(2+) in the terminal, while translocation to AZs requires Ca(2+) microdomains generated by IP3Rs. PMID: 21697360
4. This study indicated a role for patterned electrical activity and presynaptic calcium signaling, acting through CaMKII, in modulating a retrograde signal during the refinement of synaptic connections. PMID: 20920789
5. phosphorylates and regulates the eag potassium channel. PMID: 11980904
6. PS integrins function upstream of CaMKII in the development of synaptic architecture at the neuromuscular junction. PMID: 12091308
7. Regulation of neuronal excitability in Drosophila by constitutively active CaMKII. PMID: 12115891
8. This protein was characterized in transgenic animals. PMID: 12397378
9. These studies suggest two significant functions of postsynaptic CaMKII in synaptogenesis-retrograde modulation of presynaptic properties and coordinated regulation of pre- and postsynaptic maturation. PMID: 12617966
10. Retrograde control of synaptic transmission by postsynaptic CaMKII at the Drosophila neuromuscular junction. PMID: 12873383
11. CAMKII is activated by eag potassium channel PMID: 14699099
12. The sedative action of diazepam is correlated with selective down-regulation of transcripts involved in regulation of neuronal plasticity and neurotrophic responses. Most transcript changes were transient except for decrease of the CaMKIIalpha transcript PMID: 15009662
13. Morphological studies indicate that CaMKII activation in fact reduces postsynaptic sites at 8 h after hatching . This is opposite to the effect observed in larvae just after hatching. PMID: 15464287
14. a subpopulation of noncholinergic, nondopaminergic, non-GABAergic neurons can cause CaMKII-dependent reductions in initial courtship, but only cholinergic neurons enhance training-dependent suppression. PMID: 17101876
15. CaMKII-induced modulation was independent of the bone morphogenetic protein signaling that is important for retrograde control at mature larvae. PMID: 17293056

Show More

Hide All

KEGG: dme:Dmel_CG18069

STRING: 7227.FBpp0289608

UniGene: Dm.1709