PGR5 Antibody

1. The results indicate that DeltapH formation is mostly due to linear electron flow (LEF), with the contributions of PGR5- and NDH-dependent cyclic electron flow (CEF) estimated as only ~30 and ~5%, respectively. PMID: 28828567
2. PGR5-PGRL1-mediated cyclic electron transport plays a major role in DeltapH formation and induction of non-photochemical quenching. PMID: 26687812
3. PGR5 is not involved in non-photochemical reduction of plastoquinone pool and state transitions in Arabidopsis thaliana. PMID: 25792151
4. The simulation also suggested a role of the PGR5-dependent cyclic electron flow (CEF). in accelerating electron transfer in the cytochrome b6f complex. PMID: 24725611
5. Data indicate that photosystem II (PSII) yield was almost fully resistant to antimycin A in leaves accumulating endogenous levels of PtPGR5 or AtPGR5 V3K that had lysine instead of valine at the third position. PMID: 23872270
6. PROTON GRADIENT REGULATION5 (PGR5)-dependent regulation of electron transfer and proton motive force is crucial for protection of photosystem I against photodamage, which occurred particularly during the high light phases of fluctuating light cycles. PMID: 22822205
7. pgr5 mutant is affected in the redox poising of the chloroplast PMID: 17803955
8. This study suggested that a balanced PGR5 level is required for efficient regulation of the rate of antimycin A-sensitive photosystem I cyclic electron transport. PMID: 17913767
9. A complex containing PGRL1 and PGR5 is involved in the switch between linear and cyclic electron flow in Arabidopsis. PMID: 18243102
10. These results suggest that the rate of PGR5-dependent photosystem I cyclic electron transport is high enough to balance the production ratio of ATP and NADPH during steady-state photosynthesis, consistently with the pgr5 mutant phenotype. PMID: 18388110
11. PGR5 has a developmentally-regulated, conserved role in mediating cyclic electron transport. PMID: 18663471
12. PGR5 mutants show a reduction of photosynthesis and growth, which results from ATP deficiency and inactivation of PSI. PMID: 18799484

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KEGG: ath:AT2G05620

STRING: 3702.AT2G05620.1

UniGene: At.27446